How tau becomes a ‘bad guy’ that contributes to Alzheimer’s disease
The findings suggest a strategy to lower tau levels and explore the potential effects in Alzheimer’s disease and tau-induced dementias.
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Alzheimer’s Disease
Not just a ‘bad guy,’ Tau also plays a ‘good guy’ role protecting our brains
This study opens the door to potential new strategies to slow, reverse and treat neurodegenerative conditions.
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Defective serotonin 2C receptors lead to memory deficits in mice and humans
The findings not only provide new insight into the factors regulating healthy memory but also in conditions associated with memory loss, like Alzheimer’s disease.
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Fungal infection in the brain produces changes like those seen in Alzheimer’s disease
This work potentially contributes an important new piece of the puzzle regarding the development of Alzheimer’s disease.
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New Alzheimer’s disease drug shown to slow disease
The recent approval of Leqembi (lecanemab-irmb) by the U.S. Food and Drug Administration as a treatment for Alzheimer’s disease (AD) marks the first amyloid beta-directed
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Innovative machine-learning program reveals genes responsible for sex-specific differences in Alzheimer’s disease
This approach, called Evolutionary Action Machine Learning, enables researchers to probe smaller cohorts with greater accuracy.
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Supporting decision making as cognition declines
This post is a preview of a presentation for the Baruch A. Brody Lecture in Bioethics. Want to learn more about this topic? Tune into the lecture Feb.
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Interferon drives cognitive impairment in Alzheimer’s disease model
This study highlights the importance of brain inflammation and other complex events involved in the development of Alzheimer’s disease.
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Oleic acid, a key to activating the brain’s ‘fountain of youth’
Oleic acid produced in the brain is an essential regulator of the process that enables learning and memory and proper mood regulation.
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Alzheimer’s disease risk genes alter neuroprotective mechanism
New insights into factors involved in Alzheimer’s disease support that multiple alterations at the genetic and other cellular levels combine to induce the disease.
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